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Hepatic acetyl CoA links adipose tissue inflammation to hepatic insulin resistance and type 2 diabetes
AOD 9604 works by stimulating fat cell receptor pathways, not by enhancing exercise performance
It does not independently establish molecular identity, net peptide content, water content, counterion content, residual-solvent content, aggregate content, sterility, endotoxin status, or suitability for a biological application
May improve lipid processing in fat cells
The peptide simply does not interact with the receptors and pathways that stimulate IGF-1 production
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