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The proposal I want to put on the table is simple: Before prescribing a GLP-1 receptor agonist, measure the patients endogenous GLP-1 levels

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If the abovementioned is occurring, it could mean that IGF-1 administration could also lower glucose levels by suppressing hepatic gluconeogenesis, as well as improving insulin signalling in this organ, leading to IGFBP-1 suppression and overall improvement of the IGF-1/GH/insulin axis