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Based on the above results and our previous findings that inhibiting the cGAS-STING signaling pathway could suppress ferroptosis in skeletal stem cells derived from costal cartilage tissue [34], we hypothesized that GLP-1RAs might inhibit ferroptosis of chondrocytes by suppressing the activation of the cGAS-STING signaling pathway, and promote the synthesis of glycosaminoglycans and chondroitin sulfate, thereby alleviating the development of RA

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SGLT2 inhibitors: from glucose-lowering to cardiovascular benefits

The next section will explore practical approaches to preventing and managing GI AEs in clinical practice