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& Rabasa-Lhoret, R

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Some acrolein-resistant mutants showed reduced glutathione content, suggesting that additional mechanismsindependent of glutathione accumulationmay also confer acrolein resistance

This was achieved through careful selection of amino acid residues, particularly in the N-terminal region centered around the unmodified His1, and through optimization of the mid-chain and C-terminal regions to simultaneously support binding at all three receptors

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