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Nitric oxide in the pathogenesis of vascular disease
The mechanism of modulation of cyclic guanosine monophosphate accumulation by methylene blue, a putative inhibitor of soluble guanylate cyclase, was investigated in cultured rabbit pulmonary arterial smooth muscle cells
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Integrin beta 1 (ITG1), a critical cell surface protein, is actively expressed in the presence of GHK and thus may serve as a primary target for its anti-fibrotic effect [14]
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