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Similar to the statin lipid-lowering drugs mentioned earlier, other lipid-lowering drugs, such as probucol, also exhibit the capability to restore the inhibited GSH system and defend the myocardium from oxidative damage after exposure to pathological factors, for example, ISO and cyclophosphamide [242, 243]

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Published studies [1] show how selective growth hormone secretagogues (GHS) interact with the growth hormone secretagogue receptor type 1a (GHSR-1a) to trigger pulsatile GH release without uniformly activating parallel pituitary axes

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