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Although rodent models provide important tools for AD research, it is difficult for these models to fully simulate the complex pathophysiological processes of human AD because of the significant differences in brain structure, metabolic characteristics, and immune responses between humans and experimental animals (Qian et al., 2024)
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Large molecules tend to degrade significantly during digestion, reducing the amount of drug that enters circulationwhats known as bioavailability
[Crossref] Pawlak AL, Strauss E (2001) Polymorphism of the methylenetetrahydrofolate reductase gene (MTHFR) and incidence of hyperhomocysteinemia-related diseases
