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The primary mechanisms we see cited in the literature include: VEGF upregulation: BPC-157 increases vascular endothelial growth factor, which drives new capillary formation in injured tissue

7.2 Cuproptosis Cuproptosis is an oligoylated, copper-dependent form of cell death triggered by the direct binding of excess intracellular copper to lipoylated proteins involved in the tricarboxylic acid (TCA) cycle (214)